AOD-9604

​

A modified fragment of human growth hormone (hGH 176-191) developed to retain the lipolytic (fat-metabolism) activity of growth hormone while minimizing its growth-promoting effects. It is believed to stimulate lipolysis and inhibit lipogenesis by activating pathways involved in fat breakdown and metabolic regulation.

​

CJC-1295 / Ipamorelin

​

The combination of CJC-1295 and Ipamorelin is designed to stimulate endogenous growth hormone (GH) secretion through complementary mechanisms. JC-1295 acts as a growth hormone–releasing hormone (GHRH) analog, stimulating the pituitary to release GH. Ipamorelin is a ghrelin receptor (GHSR) agonist, which further promotes pulsatile GH release. Together, these peptides may enhance growth hormone and IGF-1 production, influencing tissue repair, metabolism, and anabolic signaling.

​

Ipamorelin

​

A selective growth hormone secretagogue that binds to the ghrelin receptor (GHSR-1a) in the pituitary and hypothalamus. This interaction stimulates pulsatile growth hormone release without significantly increasing cortisol or prolactin levels. It is studied for its potential role in muscle recovery, metabolic regulation, and tissue regeneration.

​

​

GHK-Cu (glycyl-L-histidyl-L-lysine copper complex)

​

A naturally occurring copper-binding tripeptide present in plasma and human tissues. It plays a role in tissue remodeling, wound healing, and extracellular matrix regulation. Research suggests it can stimulate collagen and glycosaminoglycan synthesis, promote angiogenesis, and modulate gene expression associated with skin regeneration and anti-inflammatory responses.

​

IGF-1 LR3

​

A modified form of insulin-like growth factor-1 (IGF-1) engineered for greater stability and reduced binding to IGF-binding proteins. This results in a longer biological half-life and increased receptor interaction. IGF-1 LR3 activates the IGF-1 receptor, triggering signaling pathways such as PI3K/Akt and MAPK, which regulate cell growth, protein synthesis, and tissue regeneration.

​

Lipo-C 

 

A lipotropic formulation typically containing compounds such as methionine, inositol, choline, and B vitamins that support hepatic lipid metabolism. These nutrients contribute to fat transport, methylation pathways, and mitochondrial energy production, assisting the body in the breakdown and mobilization of stored lipids.

 

NAD⁺ (Nicotinamide Adenine Dinucleotide)

​

A critical coenzyme involved in cellular redox reactions and mitochondrial energy metabolism. It serves as a key substrate for enzymes such as sirtuins, PARPs, and CD38, which regulate DNA repair, metabolic signaling, and cellular stress responses. Adequate NAD⁺ levels are essential for ATP production, metabolic efficiency, and cellular longevity mechanisms.

​

​

SLU-PP-332

​

An experimental small molecule that functions as an agonist of estrogen-related receptors (ERRα/ERRγ), nuclear receptors involved in regulating mitochondrial biogenesis and oxidative metabolism. Activation of these receptors promotes increased energy expenditure, fatty acid oxidation, and endurance-related metabolic pathways.

​

L-Carnitine

​

A naturally occurring amino acid derivative responsible for transporting long-chain fatty acids into the mitochondria for β-oxidation. This process enables the conversion of fatty acids into ATP for cellular energy production. L-Carnitine also plays roles in metabolic efficiency, exercise performance, and mitochondrial health.

​

5-Amino-1MQ

​

A small-molecule inhibitor of nicotinamide N-methyltransferase (NNMT), an enzyme involved in cellular energy metabolism and methylation processes. Inhibition of NNMT may enhance NAD⁺ availability and metabolic efficiency, potentially influencing pathways related to fat metabolism, insulin sensitivity, and energy balance.

​

Tesamorelin

​

A synthetic analog of growth hormone–releasing hormone (GHRH) that stimulates the pituitary gland to increase growth hormone secretion, which subsequently elevates IGF-1 levels. This hormonal cascade is associated with lipid metabolism regulation, visceral fat reduction, and metabolic signaling.

 

Retatrutide

​

An investigational peptide that acts as a triple agonist of the GLP-1 (glucagon-like peptide-1), GIP (glucose-dependent insulinotropic polypeptide), and glucagon receptors. Through this multi-pathway activation, it influences glucose metabolism, appetite regulation, and energy expenditure, producing significant metabolic and weight-regulation effects in clinical studies.

​

Retatrutide / Cagrilintide

​

This combination pairs Retatrutide’s multi-receptor metabolic signaling with Cagrilintide’s amylin receptor activation. The dual approach may influence satiety signaling, gastric emptying, and metabolic regulation, potentially amplifying effects on appetite control and energy balance.

​

Cagrilintide

​

A long-acting analog of amylin, a peptide hormone co-secreted with insulin by pancreatic β-cells. Amylin receptor activation contributes to satiety signaling in the brain, delayed gastric emptying, and reduced postprandial glucagon secretion, helping regulate food intake and metabolic homeostasis.

​

Tirzepatide

​

A dual agonist of the GLP-1 and GIP receptors, enhancing glucose-dependent insulin secretion while reducing glucagon release. This dual incretin signaling supports improved glycemic control, appetite regulation, and metabolic efficiency, contributing to significant effects on body weight and glucose metabolism.

​

​

SNAP-8 (Acetyl Octapeptide-3)

​

A synthetic peptide designed to modulate SNARE complex activity, which is involved in neurotransmitter release at neuromuscular junctions. By partially inhibiting neurotransmitter release responsible for muscle contraction, SNAP-8 may reduce repetitive facial muscle activity, contributing to a smoothing effect on expression lines and wrinkles.